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The pooled effect size (and 95% confidence interval [CI]) for each study was calculated as the WMD, the difference in the mean change in outcome between the weekly IER intervention and no treatment/CER intervention. Meta-analysis was pooled using a random-effect meta-analysis to provide a summary estimate. Standard deviation (SD) of the change in body weight was calculated from an imputed correlation coefficient in the study by Wadden et al. [ 28 ] based on the pre–post and change SD from the study by Arguin et al. [ 29 ]. Studies that compared two treatment groups with a single (control group) were included by combining the treatments into a single independent effect size, to prevent multi-comparisons and a unit-of-analysis error [ 30 ]. All analyses were conducted using Comprehensive Meta-Analysis (Version 3 for Windows: Biostat, Englewood, CO, USA). Heterogeneity was assessed using standard measure Cochrane's Q statistic (with p < indicating evidence of statistical heterogeneity), the I 2 statistic and T 2 [ 31 ].
by Bill Roberts - Contrary to what many would expect, this compound is actually only a weak agonist of the androgen receptor (AR), with poor binding. It follows, then, that its value must mostly come from non-AR-mediated effects. It is therefore a Class II steroid. Since it is not very effective in activating ARs, it should be stacked with a Class I steroid that is effective in this regard, such as Primobolan , Deca Durabolin , or trenbolone acetate . There is no point in stacking it with Anadrol®, which has similar activity -- one ought to simply use the more appropriate drug. With testosterone or Deca, Danabol / Dianabol is to be preferred; with Primobolan or trenbolone acetate, Anadrol® is to be preferred (though Danabol / Dianabol is still a good choice) because Anadrol® does not aromatize. For an oral-only cycle -- something I don't recommend -- Anadrol® is the better choice in my opinion for that also, at 150 mg/day (preferably divided to 3 or 6 doses.)